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Condición fitosanitaria: presente
Grupo de cultivos: Industriales
Especie hospedante: Olea europaea
Rango de hospedantes: olivo (Olea europaea), laurel de flor (Nerium oleander), ligustro (Ligustrum spp)
Etiología: Bacteria. Gram negativa. Considerada hemibiotrófica
Agente causal: Pseudomonas savastanoi (Janse 1982) Gardan et al. 1992
Taxonomía: Bacteria > Proteobacteria > Gammaproteobacteria > Pseudomonadales > Pseudomonadaceae > Pseudomonas > Pseudomonas savastanoi
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Sintomatología
Pseudomonas syringae pv. savastanoi (Smith) Young, Dye et Wilkie es una bacteria fitopatógena que causa un crecimiento excesivo de tejido (tumores o agallas), mediante la producción de ácido indol-3-acético (IAA) (Beltra, 1959) y citoquininas (Surico et al., 1976). La producción de IAA es un factor limitante en la inducción de nudos en laurel de flor (Smidt y Kosuge, 1978) y olivo (Surico et al., 1984); y la producción de IAA por cepas de pv. savastanoi de laurel, pero no por los de olivo o ligustro, se determina por plásmido (Comai et al., 1980, 1982). La cantidad de IAA producida determina la duración del período de incubación, mientras que la cantidad de citoquinina producida determina el tamaño del nudo (Surico et al., 1985). De acuerdo con el estudio de Surico et al. (1985), todas las cepas de olivo de tipo salvaje y sus mutantes exhibieron el mismo perfil de plásmido mientras que los mutantes derivados de las cepas de laurel de flor carecían de un plásmido. Esto sugiere que las cepas patógenas de laurel no llevan los determinantes genéticos para la producción de citoquininas en el plásmido que codifica para IAA.
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- Autor: Tsuji et al., 2017.
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Bibliografía
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